Surprise-surprise: Medical Research is finally catching up (a little) to my
patent written in 1996 where I showed the connection between autoimmune
disorders, decreased ATP production, and toxins.Click
here
Long-term high protein intake does not
increase oxidative stress in rats [In Process Citation]
Petzke, K. J., Elsner, A., Proll, J., Thielecke, F., Metges, C. C.
Journal of Nutrition 2000;
130: 2889-2896
The maximum dietary protein intake that
does not cause adverse effects in a healthy population is uncertain. We tested
whether a high protein intake enhances oxidative stress. Adult rats were adapted
to different casein-based diets containing either an adequate (13.8%; AP),
medium (25.7%; MP), or high (51.3%; HP) level of crude protein; a fourth group
received a HP diet but no RRR-alpha-tocopherol acetate (HP-toc). After 15 wk of
feeding, plasma protein carbonyl concentration, liver lipid peroxide levels [thiobarbituric
acid-reacting substances (TBARS)], reduced glutathione (GSH) status and leucine
kinetics ([1- (13)C]leucine) were measured. Higher concentrations of protein
carbonyls and TBARS were found in rats fed the AP and the HP-toc diets compared
with those fed the MP and HP diets (P: < 0.05). GSH concentrations in plasma
did not differ but total blood GSH concentrations were significantly (P: <
0.05) lower in rats fed the HP- toc diet compared with those fed the AP, MP and
HP diets. Liver GSH concentrations were significantly (P: < 0.01) lower in
rats fed the AP diet compared with the other groups. Rates of postabsorptive
leucine oxidation (LeuOX) and flux (Q(Leu)) were positively correlated with the
dietary protein level (for AP, MP, and HP, respectively: LeuOX, 74.9 +/- 28.5,
109 +/- 35.2, 142.3 +/- 38.4 &mgr;mol/(kg. h); Q(Leu), 425 +/- 102, 483 +/-
82, 505 +/- 80 &mgr;mol/(kg. h). Only HP-toc resulted in a significantly
greater protein breakdown (PB(Leu)) and Q(Leu). No difference was seen in
nonoxidative leucine disposal. Long-term intake of high protein diets did not
increase variables of oxidative stress, in contrast to our initial hypothesis.
An unexpected finding was that adequate protein feeding (AP) may in fact induce
oxidative stress.
ALL autoimmune disorders
are variations of the same theme.
They are NOT the body attacking itself.
The body is slowly and painfully dying from poisons exceeding its capacity
to detoxify.
The evidence abounds in the scientific literature, but is being ignored
and wrongly interpreted as
"the body attacking itself."
This is the greatest blunder of modern medicine.
There is hope, a future without
pain, and an economical path that can change your life and
put you back in control.
Donna Fezler
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